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ارداس

ارداس
دریائے کون ومکا میں!
زیتون کے تبسم کی پتیاں گراتے ہوئے
دست جنوں سے محل کے روبرو چراغ جلاتے ہوئے
اسیران وفا دل کے درواے پر دستک دے رہے تھے
ثبوت جذبات میں انگلیاں کٹ رہی تھیں
آنکھوں سے صحیفے بہہ رہے تھے
سبز لباس پہن کر۔۔۔!
محبت سنگلاخ دیواروں سے نکل رہی تھی
راہوں میں ایک گلاب بکھرتے ہوئے!
ھذا محبتً طریقتم۔۔۔و اَنا من الحقیقتم کہے جا رہا تھا
کشش قاف کی وسعتوں میں!
اک مجذوب دشتِ بیاباں کی خاک کو۔۔۔خاک شفاء کیے جا رہا تھا

مختلف مذاہب میں شہد کی افادیت، جدید سائنس کی روشنی میں تحقیقی جائزہ

This paper is the study of the fact that chief nutrition and health-relevant compounds in honey are carbohydrates, particularly glucose and fructose, which create it a superb energy resource for human. The honey comprises huge number of components in minor and trace level enzymes, proteins, minerals, vitamins and phenolic compounds, creating biological and nutritional effects like wound healing, antimicrobial, antioxidant, antiviral, antitumor and anti-inflammatory activities. The usage of honey is commonly comprised by all cultural beliefs and religious. Honey is a natural liquid mentioned in religious books and accepted by all generations, traditions and civilizations in both ancients and modern era. More than 1400 years ago, honey is described a source of healing in the Quran and it is also mentioned as one of the foods of paradise

Stratification of White Matter Hyperintensities in Young and Middle Age Adults Using Hydrogen Magnetic Resonance H-Mr Spectroscopy

Objective: The objective of this study was to stratify white matter hyperintensities on the basis of neuronal integrity in young and middle age adults by comparing the level of N-acetyl aspartate and creatine ratios in white matter hyperintensity lesions and normal appearing white matter using hydrogen magnetic resonance (H-MR) spectroscopy at 1.5 Tesla(T) Methods: Fifty consecutive patients who fulfilled the inclusion criteria aged 18-50yrs who were referred for magnetic resonance imaging(MRI) of the brain at the Radiology department of Aga Khan University Hospital over a six month period between January and June 2011 were included in the study. These patients had undergone routine MRI brain using the department protocol and were found to have white matter hyperintensities ≥5mm and no other intra-axial abnormalities. The patients underwent MR spectroscopy of the white matter hyperintensity lesions and a corresponding region of normal appearing white matter at 1.5 Tesla. Ratios of N-acetyl aspertate/creatine were compared for the white matter hyperintensity lesions and normal appearing white matter. The NAA/creatine ratios of the white matter hyperintensities and normal appearing white matter were analyzed and inferences made regarding neuronal integrity, clinical presentation and location of the white matter lesions. Results:Fifty patients, 25 male and 25 female were included in the final analysis. Thirty nine (78%) patients showed lower levels of NAA/creatine ratios in the white matter hyperintensities compared to normal appearing white matter. Eleven (22%) patients showed higher levels of NAA/creatine ratios in the white matter hyperintensities compared to normal appearing white matter. Paired T test showed a statistically significant difference between the NAA/creatine ratios in the white matter hyperintensity lesions compared to normal white matter(p value of Forty seven (94%) sampled lesions were in the subcortical and deep white matter compared to 3(6%) lesions in the periventricular location. There was no statistically significance difference in the ratios of NAA/creatine between the periventricular and deep white matter lesions. Forty seven (94%) of the study patients presented with headache . Twelve (24%) presented with dizziness. No correlation was found between the levels of NAA/creatine ratios and clinical presentation. Conclusion: This study demonstrates that white matter hyperintensities in young and middle age adults are associated with actual neuronal destruction. These patients should be investigated to define the aetiology and if possible interventions to reverse or prevent further neuronal damage.
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